Activation of alternative NF-κB signaling during recovery of disuse-induced loss of muscle oxidative phenotype. [electronic resource]
Producer: 20140527Description: E615-26 p. digitalISSN:- 1522-1555
- Animals
- Biomarkers -- metabolism
- DNA-Binding Proteins -- biosynthesis
- Disease Models, Animal
- Gene Expression Regulation
- High Mobility Group Proteins -- biosynthesis
- Hindlimb Suspension
- Immobilization -- adverse effects
- Mice
- Mice, Inbred C57BL
- Mitochondrial Proteins -- biosynthesis
- Muscle, Skeletal -- metabolism
- Muscular Disorders, Atrophic -- etiology
- Myosin Heavy Chains -- biosynthesis
- NF-kappa B -- agonists
- Oxidative Phosphorylation
- Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
- Random Allocation
- Receptors, Estrogen -- biosynthesis
- Signal Transduction
- Transcription Factors -- biosynthesis
- Weight-Bearing
- ERRalpha Estrogen-Related Receptor
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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