Transgenic expression of neuronal dystonin isoform 2 partially rescues the disease phenotype of the dystonia musculorum mouse model of hereditary sensory autonomic neuropathy VI. [electronic resource]
Producer: 20141203Description: 2694-710 p. digitalISSN:- 1460-2083
- Animals
- Carrier Proteins -- genetics
- Cells, Cultured
- Cytoskeletal Proteins -- genetics
- Disease Models, Animal
- Dystonia Musculorum Deformans -- genetics
- Dystonin
- Ganglia, Spinal -- pathology
- Hereditary Sensory and Autonomic Neuropathies -- genetics
- Humans
- Intracellular Membranes -- metabolism
- Mice, Inbred C57BL
- Mice, Transgenic
- Microtubules -- metabolism
- Muscle Spindles -- metabolism
- Nerve Fibers, Myelinated -- metabolism
- Nerve Tissue Proteins -- genetics
- Neuromuscular Junction -- metabolism
- Phenotype
- Proprioception
- Sensory Receptor Cells -- pathology
- Transgenes
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.