Inhibition of excitatory synaptic transmission in hippocampal neurons by levetiracetam involves Zn²⁺-dependent GABA type A receptor-mediated presynaptic modulation. [electronic resource]
Producer: 20140303Description: 246-59 p. digitalISSN:- 1521-0103
- Action Potentials -- drug effects
- Animals
- Anticonvulsants -- pharmacology
- CA3 Region, Hippocampal -- cytology
- Cells, Cultured
- Chelating Agents -- pharmacology
- Evoked Potentials -- drug effects
- Female
- GABA-A Receptor Agonists -- pharmacology
- GABA-A Receptor Antagonists -- pharmacology
- GABAergic Neurons -- cytology
- Levetiracetam
- Male
- Mossy Fibers, Hippocampal -- drug effects
- Nerve Tissue Proteins -- agonists
- Neural Inhibition -- drug effects
- Piracetam -- analogs & derivatives
- Presynaptic Terminals -- drug effects
- Rats
- Receptors, GABA-A -- chemistry
- Zinc -- chemistry
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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