Hypercholesterolemic myocardium is vulnerable to ischemia-reperfusion injury and refractory to sevoflurane-induced protection. [electronic resource]
Producer: 20140522Description: e76652 p. digitalISSN:- 1932-6203
- Anesthetics, Inhalation -- administration & dosage
- Animals
- Apoptosis -- drug effects
- Cardiotonic Agents -- administration & dosage
- Coronary Artery Disease -- etiology
- Diet
- Glycogen Synthase Kinase 3 -- antagonists & inhibitors
- Glycogen Synthase Kinase 3 beta
- Heart Ventricles -- drug effects
- Hemodynamics -- drug effects
- Hypercholesterolemia -- complications
- Ischemic Postconditioning
- Male
- Methyl Ethers -- administration & dosage
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Muscular Dystrophies, Limb-Girdle -- metabolism
- Myocardial Reperfusion Injury -- drug therapy
- Myocardium -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Proto-Oncogene Proteins c-akt -- metabolism
- Rats
- Sevoflurane
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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