Loss of TAK1 increases cell traction force in a ROS-dependent manner to drive epithelial-mesenchymal transition of cancer cells. [electronic resource]
Producer: 20140312Description: e848 p. digitalISSN:- 2041-4889
- Animals
- Biomechanical Phenomena -- drug effects
- Cell Line, Tumor
- Cell Membrane -- drug effects
- Epithelial-Mesenchymal Transition -- drug effects
- HEK293 Cells
- Humans
- Integrin beta1 -- metabolism
- Integrin beta3 -- metabolism
- MAP Kinase Kinase Kinases -- deficiency
- Mice
- Models, Biological
- Neoplasm Invasiveness
- Neoplasms, Squamous Cell -- enzymology
- Oxidation-Reduction -- drug effects
- Reactive Oxygen Species -- metabolism
- Signal Transduction -- drug effects
- Skin Neoplasms -- enzymology
- Smad Proteins -- metabolism
- Transforming Growth Factor beta1 -- pharmacology
- rac1 GTP-Binding Protein -- metabolism
- rho GTP-Binding Proteins -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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