Luteolin induces growth arrest in colon cancer cells through involvement of Wnt/β-catenin/GSK-3β signaling. [electronic resource]
Producer: 20131210Description: 131-9 p. digitalISSN:- 2162-6537
- Adenocarcinoma -- drug therapy
- Antineoplastic Agents, Phytogenic -- pharmacology
- Apoptosis -- drug effects
- Caspase 3 -- metabolism
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Colonic Neoplasms -- drug therapy
- Cyclin D1 -- genetics
- Dose-Response Relationship, Drug
- G2 Phase Cell Cycle Checkpoints -- drug effects
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Humans
- Luteolin -- pharmacology
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Signal Transduction -- drug effects
- Wnt Signaling Pathway -- drug effects
- bcl-2-Associated X Protein -- metabolism
- beta Catenin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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