Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a. [electronic resource]
Producer: 20131210Description: 4308-16 p. digitalISSN:- 1550-6606
- Aminoquinolines
- Butadienes -- pharmacology
- Cells, Cultured
- Complement C5a -- metabolism
- Extracellular Signal-Regulated MAP Kinases -- antagonists & inhibitors
- GTP-Binding Protein alpha Subunits, Gi-Go -- antagonists & inhibitors
- Granulocyte-Macrophage Colony-Stimulating Factor
- Humans
- Imiquimod
- Inflammation -- chemically induced
- Interleukin-10 -- biosynthesis
- Interleukin-6 -- metabolism
- JNK Mitogen-Activated Protein Kinases -- metabolism
- Lipopolysaccharides
- MAP Kinase Signaling System -- immunology
- Macrophage Colony-Stimulating Factor
- Macrophages -- metabolism
- Mitogen-Activated Protein Kinase Kinases -- antagonists & inhibitors
- Monocytes -- metabolism
- Nitriles -- pharmacology
- Pertussis Toxin -- pharmacology
- Phosphatidylinositol 3-Kinases -- metabolism
- Poly I-C
- Proto-Oncogene Proteins c-raf -- metabolism
- Receptor, Anaphylatoxin C5a
- Receptors, Complement -- metabolism
- Salmonella typhimurium -- immunology
- Toll-Like Receptor 4 -- metabolism
- Tumor Necrosis Factors -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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