Lipopolysaccharide-induced cross-tolerance against renal ischemia-reperfusion injury is mediated by hypoxia-inducible factor-2α-regulated nitric oxide production. [electronic resource]
Producer: 20140929Description: 276-88 p. digitalISSN:- 1523-1755
- Acute Kidney Injury -- genetics
- Animals
- Basic Helix-Loop-Helix Transcription Factors -- deficiency
- Bone Marrow Transplantation
- Cells, Cultured
- Cytoprotection
- Disease Models, Animal
- Endothelial Cells -- drug effects
- Enzyme Inhibitors -- pharmacology
- Human Umbilical Vein Endothelial Cells -- drug effects
- Humans
- Hypoxia-Inducible Factor 1, alpha Subunit -- genetics
- Kidney -- blood supply
- Lipopolysaccharides -- pharmacology
- Male
- Mice
- Mice, Knockout
- Microcirculation -- drug effects
- NF-kappa B -- metabolism
- Nitric Oxide -- metabolism
- Nitric Oxide Donors -- pharmacology
- Nitric Oxide Synthase Type II -- antagonists & inhibitors
- Nitric Oxide Synthase Type III -- antagonists & inhibitors
- RNA Interference
- Renal Circulation -- drug effects
- Reperfusion Injury -- genetics
- Signal Transduction
- Time Factors
- Transfection
- Warm Ischemia
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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