Activated microglia mediate synapse loss and short-term memory deficits in a mouse model of transthyretin-related oculoleptomeningeal amyloidosis. [electronic resource]
Producer: 20140312Description: e789 p. digitalISSN:- 2041-4889
- Amyloid
- Amyloid Neuropathies, Familial -- complications
- Animals
- Brain -- metabolism
- Cell Death -- drug effects
- Cell Nucleus -- metabolism
- Cells, Cultured
- Culture Media, Conditioned -- pharmacology
- Disease Models, Animal
- Endocytosis
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Inflammation Mediators -- metabolism
- Interleukin-6 -- metabolism
- Memory Disorders -- complications
- Memory, Short-Term -- drug effects
- Mice
- Microglia -- drug effects
- Minocycline -- pharmacology
- Mutation -- genetics
- NF-kappa B -- metabolism
- Neurons -- drug effects
- Phosphorylation
- Prealbumin -- metabolism
- Protein Transport
- Proto-Oncogene Proteins c-akt -- metabolism
- Synapses -- drug effects
- Tumor Necrosis Factor-alpha -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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