Bioenergetic deficits in peripheral nerve sensory axons during chemotherapy-induced neuropathic pain resulting from peroxynitrite-mediated post-translational nitration of mitochondrial superoxide dismutase. [electronic resource]
Producer: 20140528Description: 2432-2440 p. digitalISSN:- 1872-6623
- Adenosine Triphosphate -- metabolism
- Animals
- Antineoplastic Agents -- adverse effects
- Antineoplastic Agents, Phytogenic -- pharmacology
- Axons -- physiology
- Boronic Acids -- pharmacology
- Bortezomib
- Energy Metabolism -- physiology
- Hyperalgesia -- drug therapy
- Male
- Mitochondria -- drug effects
- Neoplasm Transplantation
- Neuralgia -- chemically induced
- Organoplatinum Compounds -- pharmacology
- Oxaliplatin
- Paclitaxel -- pharmacology
- Peripheral Nerves -- physiology
- Peroxynitrous Acid -- physiology
- Physical Stimulation
- Protein Processing, Post-Translational -- physiology
- Pyrazines -- pharmacology
- Rats
- Rats, Sprague-Dawley
- Sensory Receptor Cells -- physiology
- Superoxide Dismutase -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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