SPARC regulates microgliosis and functional recovery following cortical ischemia. [electronic resource]
Producer: 20130426Description: 4468-81 p. digitalISSN:- 1529-2401
- Age Factors
- Animals
- Animals, Newborn
- Brain Infarction -- etiology
- Brain Ischemia -- complications
- CX3C Chemokine Receptor 1
- Calcium-Binding Proteins -- metabolism
- Cell Count
- Cell Movement -- drug effects
- Cell Proliferation -- drug effects
- Cell Size
- Cells, Cultured
- Cerebral Cortex -- pathology
- Disease Models, Animal
- Excitatory Amino Acid Agonists -- toxicity
- Female
- Forelimb -- physiopathology
- Galectin 3 -- metabolism
- Gene Expression Regulation, Developmental -- drug effects
- Genotype
- Glial Fibrillary Acidic Protein -- metabolism
- Gliosis -- etiology
- Glycoproteins -- deficiency
- Green Fluorescent Proteins -- genetics
- Intracranial Thrombosis -- complications
- Lectins -- metabolism
- Male
- Mice
- Mice, Transgenic
- Microfilament Proteins -- metabolism
- Microglia -- drug effects
- Motor Skills -- drug effects
- Mutation -- genetics
- N-Methylaspartate -- toxicity
- Olfactory Bulb -- injuries
- Osteonectin
- Proliferating Cell Nuclear Antigen -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Receptors, Chemokine -- genetics
- Recovery of Function -- physiology
- Time Factors
- Tumor Suppressor Proteins -- deficiency
- NF-kappaB-Inducing Kinase
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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