Cancer stem-like cell properties are regulated by EGFR/AKT/β-catenin signaling and preferentially inhibited by gefitinib in nasopharyngeal carcinoma. [electronic resource]
Producer: 20130618Description: 2027-41 p. digitalISSN:- 1742-4658
- Animals
- Antineoplastic Agents -- pharmacology
- Carcinoma -- drug therapy
- Cell Line, Tumor
- Cell Proliferation
- Cell Survival -- drug effects
- Cell Transformation, Neoplastic -- metabolism
- Cisplatin -- pharmacology
- ErbB Receptors -- antagonists & inhibitors
- Gefitinib
- Gene Knockdown Techniques
- Homeodomain Proteins -- metabolism
- Humans
- Mice
- Mice, Inbred BALB C
- Mice, Nude
- Nanog Homeobox Protein
- Nasopharyngeal Carcinoma
- Nasopharyngeal Neoplasms -- drug therapy
- Neoplasm Transplantation
- Neoplastic Stem Cells -- drug effects
- Proto-Oncogene Proteins c-akt -- metabolism
- Quinazolines -- pharmacology
- Side-Population Cells -- drug effects
- Signal Transduction
- Spheroids, Cellular -- metabolism
- Xenograft Model Antitumor Assays
- beta Catenin -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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