Impaired mitochondrial biogenesis due to dysfunctional adiponectin-AMPK-PGC-1α signaling contributing to increased vulnerability in diabetic heart. [electronic resource]
Producer: 20130816Description: 329 p. digitalISSN:- 1435-1803
- AMP-Activated Protein Kinases -- metabolism
- Acetylation
- Adenosine Triphosphate -- metabolism
- Adiponectin -- deficiency
- Animals
- Animals, Newborn
- Cells, Cultured
- DNA, Mitochondrial -- metabolism
- DNA-Binding Proteins -- metabolism
- Diabetes Complications -- enzymology
- Disease Models, Animal
- Electron Transport Chain Complex Proteins -- metabolism
- Energy Metabolism
- High Mobility Group Proteins -- metabolism
- Leptin -- deficiency
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Microscopy, Electron
- Mitochondria, Heart -- enzymology
- Mitochondrial Swelling
- Mitochondrial Turnover
- Myocardial Infarction -- enzymology
- Myocardium -- enzymology
- Nuclear Respiratory Factor 1 -- metabolism
- Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
- RNA Interference
- RNA, Messenger -- metabolism
- RNA-Binding Proteins -- metabolism
- Rats
- Rats, Sprague-Dawley
- Signal Transduction
- Time Factors
- Trans-Activators -- genetics
- Transcription Factors -- metabolism
- Transfection
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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