FGF-2 and FGF-16 protect isolated perfused mouse hearts from acute doxorubicin-induced contractile dysfunction. [electronic resource]
Producer: 20140402Description: 244-53 p. digitalISSN:- 1559-0259
- Animals
- Anti-Arrhythmia Agents
- Antibiotics, Antineoplastic -- antagonists & inhibitors
- Blood Pressure -- drug effects
- Cardiotonic Agents
- Coronary Circulation -- drug effects
- Doxorubicin -- antagonists & inhibitors
- Enzyme Activation -- drug effects
- Fibroblast Growth Factor 2 -- biosynthesis
- Fibroblast Growth Factors -- biosynthesis
- Heart Diseases -- chemically induced
- In Vitro Techniques
- L-Lactate Dehydrogenase -- metabolism
- Mice
- Myocardial Contraction -- drug effects
- Myocardial Reperfusion Injury -- physiopathology
- Protein Kinase C -- metabolism
- Recombinant Proteins -- pharmacology
- Reperfusion Injury -- prevention & control
- Ventricular Function, Left -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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