Apoptosis induced by trimethyltin chloride in human neuroblastoma cells SY5Y is regulated by a balance and cross-talk between NF-κB and MAPKs signaling pathways. [electronic resource]
Producer: 20131017Description: 1273-85 p. digitalISSN:- 1432-0738
- Apoptosis -- drug effects
- Cell Line, Tumor
- Cell Survival -- drug effects
- Dose-Response Relationship, Drug
- Enzyme Activation
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Humans
- I-kappa B Proteins -- metabolism
- JNK Mitogen-Activated Protein Kinases -- metabolism
- MAP Kinase Signaling System -- drug effects
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Neuroblastoma -- enzymology
- Neurons -- drug effects
- Phosphorylation
- Proteasome Endopeptidase Complex -- metabolism
- Proteasome Inhibitors -- pharmacology
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Time Factors
- Transcription Factor RelA -- metabolism
- Trimethyltin Compounds -- toxicity
- X-Linked Inhibitor of Apoptosis Protein -- metabolism
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article
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