CD36, but not G2A, modulates efferocytosis, inflammation, and fibrosis following bleomycin-induced lung injury. [electronic resource]
Producer: 20130930Description: 1114-23 p. digitalISSN:- 1539-7262
- Animals
- Apoptosis -- drug effects
- Bleomycin -- toxicity
- CD36 Antigens -- genetics
- Cell Cycle Proteins -- genetics
- Fluorescent Antibody Technique
- Immunohistochemistry
- In Situ Nick-End Labeling
- Inflammation -- genetics
- Lung Injury -- chemically induced
- Mice
- Mice, Knockout
- Receptors, G-Protein-Coupled -- genetics
- Spectrometry, Mass, Electrospray Ionization
- Tandem Mass Spectrometry
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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