Cyclosporine A normalizes mitochondrial coupling, reactive oxygen species production, and inflammation and partially restores skeletal muscle maximal oxidative capacity in experimental aortic cross-clamping. [electronic resource]
Producer: 20130522Description: 1100-1108.e2 p. digitalISSN:- 1097-6809
- Animals
- Aorta -- physiopathology
- Blotting, Western
- Calcium -- metabolism
- Constriction
- Peptidyl-Prolyl Isomerase F
- Cyclophilins -- metabolism
- Cyclosporine -- administration & dosage
- Disease Models, Animal
- Electron Transport Chain Complex Proteins -- metabolism
- Energy Metabolism -- drug effects
- Immunohistochemistry
- Inflammation -- metabolism
- Injections, Intraperitoneal
- Macrophages -- drug effects
- Male
- Mitochondria, Heart -- drug effects
- Mitochondria, Muscle -- drug effects
- Mitochondrial Membrane Transport Proteins -- antagonists & inhibitors
- Mitochondrial Permeability Transition Pore
- Muscle, Skeletal -- blood supply
- Oxidative Stress -- drug effects
- Rats
- Rats, Wistar
- Reactive Oxygen Species -- metabolism
- Reperfusion Injury -- metabolism
- Time Factors
No physical items for this record
Publication Type: Journal Article
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