Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. [electronic resource]
Producer: 20130509Description: C508-18 p. digitalISSN:- 1522-1563
- Animals
- Apoptosis -- drug effects
- Calcium-Binding Proteins -- metabolism
- Cells, Cultured
- Colforsin -- pharmacology
- Diabetic Cardiomyopathies -- metabolism
- Endoplasmic Reticulum Stress -- drug effects
- Enzyme Activation
- Exenatide
- Glucagon-Like Peptide-1 Receptor
- Glucose -- metabolism
- HSP70 Heat-Shock Proteins
- Heat-Shock Proteins -- metabolism
- Hydrogen Peroxide -- pharmacology
- Hyperglycemia -- metabolism
- Hypoglycemic Agents -- pharmacology
- Isoquinolines -- pharmacology
- Membrane Proteins
- Myocytes, Cardiac -- physiology
- Oxidative Stress -- drug effects
- Peptide Fragments -- pharmacology
- Peptides -- pharmacology
- Phosphorylation -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- RNA, Messenger -- biosynthesis
- Rats
- Receptors, Glucagon -- agonists
- Sarcoplasmic Reticulum Calcium-Transporting ATPases -- antagonists & inhibitors
- Sulfonamides -- pharmacology
- Thapsigargin -- pharmacology
- Transcription Factor CHOP -- biosynthesis
- Tunicamycin -- pharmacology
- Unfolded Protein Response
- Venoms -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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