Loss of CDKN2B promotes p53-dependent smooth muscle cell apoptosis and aneurysm formation. [electronic resource]
Producer: 20130212Description: e1-e10 p. digitalISSN:- 1524-4636
- Adolescent
- Adult
- Aged
- Animals
- Aorta, Abdominal -- metabolism
- Aortic Aneurysm, Abdominal -- chemically induced
- Apoptosis -- drug effects
- Benzothiazoles -- pharmacology
- Bone Marrow Transplantation
- Carotid Arteries -- metabolism
- Carotid Artery Diseases -- genetics
- Case-Control Studies
- Cell Movement
- Cell Proliferation
- Cells, Cultured
- Child
- Child, Preschool
- Cyclin-Dependent Kinase Inhibitor p15 -- deficiency
- Disease Models, Animal
- Gene Expression Regulation
- Genotype
- Humans
- Infant
- Infant, Newborn
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Middle Aged
- Muscle, Smooth, Vascular -- drug effects
- Neointima
- Pancreatic Elastase
- Phenotype
- Proto-Oncogene Proteins c-mdm2 -- metabolism
- RNA Interference
- Signal Transduction
- Time Factors
- Toluene -- analogs & derivatives
- Transfection
- Tumor Suppressor Protein p53 -- antagonists & inhibitors
- Young Adult
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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