Activation of canonical wnt pathway promotes differentiation of mouse bone marrow-derived MSCs into type II alveolar epithelial cells, confers resistance to oxidative stress, and promotes their migration to injured lung tissue in vitro. [electronic resource]
Producer: 20130418Description: 1270-83 p. digitalISSN:- 1097-4652
- Acute Lung Injury -- metabolism
- Alveolar Epithelial Cells -- drug effects
- Animals
- Biomarkers -- metabolism
- Bone Marrow Cells -- drug effects
- Cell Differentiation -- drug effects
- Cell Movement
- Cell Proliferation
- Cells, Cultured
- Coculture Techniques
- Culture Media, Conditioned -- metabolism
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Hydrogen Peroxide -- pharmacology
- Lithium Chloride -- pharmacology
- Male
- Mesenchymal Stem Cell Transplantation
- Mesenchymal Stem Cells -- drug effects
- Mice
- Mice, Inbred C57BL
- Oxidants -- pharmacology
- Oxidative Stress -- drug effects
- Re-Epithelialization
- Time Factors
- Tissue Culture Techniques
- Wnt Signaling Pathway -- drug effects
- Wnt3A Protein -- metabolism
- beta Catenin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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