c-Fms signaling mediates neurofibromatosis Type-1 osteoclast gain-in-functions. [electronic resource]
Producer: 20130422Description: e46900 p. digitalISSN:- 1932-6203
- Animals
- Bone Resorption -- drug therapy
- Cell Adhesion -- drug effects
- Cell Movement -- drug effects
- Cells, Cultured
- Enzyme Inhibitors -- therapeutic use
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Female
- Gene Deletion
- Humans
- Macrophage Colony-Stimulating Factor -- metabolism
- Mice
- Mice, Inbred C57BL
- Neurofibromin 1 -- genetics
- Osteoclasts -- cytology
- Osteoporosis -- drug therapy
- Receptor, Macrophage Colony-Stimulating Factor -- antagonists & inhibitors
- Ribosomal Protein S6 Kinases, 90-kDa -- metabolism
- Signal Transduction
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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