Presenilin-1 adopts pathogenic conformation in normal aging and in sporadic Alzheimer's disease. [electronic resource]
Producer: 20130625Description: 187-99 p. digitalISSN:- 1432-0533
- Aged, 80 and over
- Aging -- physiology
- Alzheimer Disease -- genetics
- Amyloid beta-Peptides -- chemistry
- Animals
- Cells, Cultured
- Female
- Frontotemporal Lobar Degeneration -- genetics
- Humans
- Immunohistochemistry
- Male
- Mice
- Mice, Transgenic
- Microscopy, Fluorescence
- Neurons -- metabolism
- Oxidative Stress
- Peptide Fragments -- metabolism
- Plaque, Amyloid -- genetics
- Presenilin-1 -- chemistry
- Protein Conformation
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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