Atypical activation of the unfolded protein response in cystic fibrosis airway cells contributes to p38 MAPK-mediated innate immune responses. [electronic resource]
Producer: 20130118Description: 5467-75 p. digitalISSN:- 1550-6606
- Cells, Cultured
- Cinnamates -- pharmacology
- Cystic Fibrosis -- complications
- Endoplasmic Reticulum -- drug effects
- Endoplasmic Reticulum Stress -- drug effects
- Epithelial Cells -- immunology
- Eukaryotic Initiation Factor-2 -- genetics
- Flagellin -- immunology
- Gene Expression Profiling
- Gene Expression Regulation -- drug effects
- Humans
- Immunity, Innate -- drug effects
- Interleukin-6 -- biosynthesis
- Lung -- immunology
- Pneumonia -- complications
- Pseudomonas aeruginosa -- immunology
- Signal Transduction -- drug effects
- Thiourea -- analogs & derivatives
- Unfolded Protein Response -- drug effects
- eIF-2 Kinase -- genetics
- p38 Mitogen-Activated Protein Kinases -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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