Restoration of dopamine release deficits during object recognition memory acquisition attenuates cognitive impairment in a triple transgenic mice model of Alzheimer's disease. [electronic resource]
Producer: 20130222Description: 453-60 p. digitalISSN:- 1549-5485
- Age Factors
- Alzheimer Disease -- complications
- Amyloid beta-Peptides -- metabolism
- Amyloid beta-Protein Precursor -- genetics
- Animals
- Brain -- drug effects
- Cognition Disorders -- drug therapy
- Disease Models, Animal
- Dopamine -- metabolism
- Dopamine Uptake Inhibitors -- therapeutic use
- Exploratory Behavior -- drug effects
- Glutamic Acid -- metabolism
- Humans
- Male
- Memory Disorders -- drug therapy
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Microdialysis
- Mutation -- genetics
- Nomifensine -- therapeutic use
- Norepinephrine -- metabolism
- Photic Stimulation
- Presenilin-1 -- genetics
- Recognition, Psychology -- drug effects
- Statistics, Nonparametric
- tau Proteins -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.