Disruption of Nox2 and TNFRp55/p75 eliminates cardioprotection induced by anisomycin. [electronic resource]
Producer: 20130117Description: H1263-72 p. digitalISSN:- 1522-1539
- Animals
- Anisomycin -- administration & dosage
- Disease Models, Animal
- Enzyme Activation
- Enzyme Activators -- administration & dosage
- Injections, Intraperitoneal
- Male
- Membrane Glycoproteins -- deficiency
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardial Infarction -- drug therapy
- Myocardial Reperfusion Injury -- drug therapy
- Myocardium -- metabolism
- NADPH Oxidase 2
- NADPH Oxidases -- deficiency
- Protein Kinase Inhibitors -- pharmacology
- Receptors, Tumor Necrosis Factor, Type I -- deficiency
- Receptors, Tumor Necrosis Factor, Type II -- deficiency
- Signal Transduction -- drug effects
- Superoxides -- metabolism
- Time Factors
- Tumor Necrosis Factor-alpha -- metabolism
- Ventricular Function, Left -- drug effects
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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