B-lymphopoiesis is stopped by mobilizing doses of G-CSF and is rescued by overexpression of the anti-apoptotic protein Bcl2. [electronic resource]
Producer: 20131126Description: 325-33 p. digitalISSN:- 1592-8721
- Animals
- B-Lymphocyte Subsets -- drug effects
- Benzylamines
- Bone Marrow -- drug effects
- Cyclams
- Cyclophosphamide -- pharmacology
- Gene Expression
- Granulocyte Colony-Stimulating Factor -- administration & dosage
- Hematopoietic Stem Cell Mobilization
- Heterocyclic Compounds -- pharmacology
- Lymphopoiesis -- drug effects
- Male
- Mice
- Precursor Cells, B-Lymphoid -- drug effects
- Proto-Oncogene Proteins c-bcl-2 -- genetics
- Spleen -- drug effects
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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