Reperfusion-triggered stress protein response in the myocardium is blocked by post-conditioning. Systems biology pathway analysis highlights the key role of the canonical aryl-hydrocarbon receptor pathway. [electronic resource]
Producer: 20140211Description: 2082-93 p. digitalISSN:- 1522-9645
- Animals
- Apoptosis -- physiology
- Aryl Hydrocarbon Receptor Nuclear Translocator -- metabolism
- Caspase 8 -- metabolism
- Coronary Vessels -- pathology
- Deoxyuracil Nucleotides -- metabolism
- Echocardiography
- Heat-Shock Proteins -- physiology
- Ischemic Postconditioning -- methods
- Myocardial Contraction -- physiology
- Myocardial Reperfusion Injury -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Proteome -- metabolism
- Proteomics
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Random Allocation
- Receptors, Aryl Hydrocarbon -- metabolism
- Signal Transduction -- physiology
- Sus scrofa
- Systems Biology
- TOR Serine-Threonine Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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