Intranasal administration of aTf protects and repairs the neonatal white matter after a cerebral hypoxic-ischemic event. [electronic resource]
Producer: 20130115Description: 1540-54 p. digitalISSN:- 1098-1136
- 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase -- genetics
- Administration, Intranasal
- Age Factors
- Animals
- Animals, Newborn
- Antigens -- metabolism
- Apoproteins -- administration & dosage
- Autophagy-Related Proteins
- Brain Injuries -- etiology
- Bromodeoxyuridine -- metabolism
- Caspase 3 -- metabolism
- Cell Death -- drug effects
- Cell Proliferation -- drug effects
- Cells, Cultured
- Cerebral Cortex -- cytology
- Colchicine -- pharmacology
- Corpus Callosum -- drug effects
- Cytochalasin B -- pharmacology
- Female
- Gene Expression Regulation -- drug effects
- Green Fluorescent Proteins -- genetics
- Humans
- Hypoxia -- drug therapy
- Hypoxia-Ischemia, Brain -- complications
- Intermediate Filament Proteins -- metabolism
- Intracellular Signaling Peptides and Proteins -- metabolism
- Lateral Ventricles -- drug effects
- Male
- Mice
- Mice, Transgenic
- Myelin Basic Protein -- metabolism
- Nerve Fibers, Myelinated -- drug effects
- Nerve Tissue Proteins -- metabolism
- Nestin
- Neurogenesis -- drug effects
- Neuroprotective Agents -- administration & dosage
- Oligodendroglia -- drug effects
- Platelet-Derived Growth Factor -- metabolism
- Proteoglycans -- metabolism
- SOXB1 Transcription Factors -- metabolism
- Time Factors
- Transferrin -- administration & dosage
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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