Respiratory syncytial virus infection augments NOD2 signaling in an IFN-β-dependent manner in human primary cells. [electronic resource]
Producer: 20130111Description: 2727-35 p. digitalISSN:- 1521-4141
- Acetylmuramyl-Alanyl-Isoglutamine -- immunology
- Cells, Cultured
- Crohn Disease -- immunology
- DEAD Box Protein 58
- DEAD-box RNA Helicases -- genetics
- Humans
- Infant
- Infant, Newborn
- Inflammation Mediators -- metabolism
- Interferon-beta -- metabolism
- Mutation -- genetics
- Nod2 Signaling Adaptor Protein -- genetics
- Primary Cell Culture
- RNA, Viral -- metabolism
- Receptors, Immunologic
- Respiratory Syncytial Virus Infections -- immunology
- Respiratory Syncytial Viruses -- physiology
- Signal Transduction
- Toll-Like Receptor 3 -- genetics
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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