Phosphoinositide-3-kinase/akt - dependent signaling is required for maintenance of [Ca(2+)](i), I(Ca), and Ca(2+) transients in HL-1 cardiomyocytes. [electronic resource]
Producer: 20130104Description: 59 p. digitalISSN:- 1423-0127
- Animals
- Calcium -- metabolism
- Electrophysiology
- Excitation Contraction Coupling
- Fura-2 -- analysis
- Mice
- Morpholines -- pharmacology
- Myocardial Contraction -- genetics
- Myocardium -- pathology
- Myocytes, Cardiac -- metabolism
- Patch-Clamp Techniques
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation
- Proto-Oncogene Proteins c-akt -- metabolism
- Pyrimidinones -- pharmacology
- Signal Transduction -- drug effects
- Thiazolidinediones -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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