Cardiac vulnerability to ischemia/reperfusion injury drastically increases in late pregnancy. [electronic resource]
Producer: 20120827Description: 271 p. digitalISSN:- 1435-1803
- Animals
- Calcium -- metabolism
- Disease Models, Animal
- Disease Susceptibility
- Female
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Mice
- Mice, Inbred C57BL
- Mitochondria, Heart -- metabolism
- Mitochondrial Membrane Transport Proteins -- metabolism
- Mitochondrial Permeability Transition Pore
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Myocardial Infarction -- etiology
- Myocardial Reperfusion Injury -- etiology
- Myocardium -- metabolism
- Oxidative Stress
- Phosphorylation
- Pregnancy
- Pregnancy Complications, Cardiovascular -- etiology
- Proto-Oncogene Proteins c-akt -- metabolism
- Rats
- Rats, Sprague-Dawley
- Reactive Oxygen Species -- metabolism
- Recovery of Function
- STAT3 Transcription Factor -- metabolism
- Signal Transduction
- Time Factors
- Ventricular Function, Left
- Ventricular Pressure
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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