Pathogenesis of NOD diabetes is initiated by reactivity to the insulin B chain 9-23 epitope and involves functional epitope spreading. [electronic resource]
Producer: 20130425Description: 347-53 p. digitalISSN:- 1095-9157
- Animals
- Autoantigens -- genetics
- Autoimmunity
- Cell Movement
- Cross-Linking Reagents -- chemistry
- Diabetes Mellitus, Type 1 -- genetics
- Disease Progression
- Epitopes -- genetics
- Female
- Glucose-6-Phosphatase -- genetics
- Glutamate Decarboxylase -- genetics
- Immune Tolerance
- Injections, Intravenous
- Insulin -- genetics
- Insulin-Secreting Cells -- immunology
- Mice
- Mice, Inbred NOD
- Peptide Fragments -- genetics
- Proteins -- genetics
- Spleen -- immunology
- T-Lymphocytes, Regulatory -- immunology
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.