Yohimbine depresses excitatory transmission in BNST and impairs extinction of cocaine place preference through orexin-dependent, norepinephrine-independent processes. [electronic resource]
Producer: 20130109Description: 2253-66 p. digitalISSN:- 1740-634X
- Adrenergic alpha-1 Receptor Antagonists -- pharmacology
- Adrenergic alpha-2 Receptor Antagonists -- pharmacology
- Animals
- Behavior, Animal -- drug effects
- Cocaine -- pharmacology
- Drug-Seeking Behavior -- drug effects
- Extinction, Psychological -- drug effects
- Intracellular Signaling Peptides and Proteins -- pharmacology
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Morpholines -- pharmacology
- Neuropeptides -- pharmacology
- Neurotransmitter Agents -- pharmacology
- Norepinephrine -- physiology
- Norepinephrine Plasma Membrane Transport Proteins -- antagonists & inhibitors
- Orexin Receptors
- Orexins
- Patch-Clamp Techniques
- Reboxetine
- Receptors, Adrenergic, alpha-2 -- genetics
- Receptors, Corticotropin-Releasing Hormone -- antagonists & inhibitors
- Receptors, Neuropeptide -- antagonists & inhibitors
- Reward
- Septal Nuclei -- drug effects
- Synaptic Transmission -- drug effects
- Yohimbine -- pharmacology
- CRF Receptor, Type 1
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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