CD147 induces UPR to inhibit apoptosis and chemosensitivity by increasing the transcription of Bip in hepatocellular carcinoma. [electronic resource]
Producer: 20130304Description: 1779-90 p. digitalISSN:- 1476-5403
- Animals
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Basigin -- chemistry
- Biomarkers, Tumor -- metabolism
- Carcinoma, Hepatocellular -- drug therapy
- Cell Line, Tumor
- Doxorubicin -- pharmacology
- Endoplasmic Reticulum Chaperone BiP
- Endoplasmic Reticulum Stress
- Focal Adhesion Kinase 1 -- metabolism
- HEK293 Cells
- Heat-Shock Proteins -- genetics
- Hep G2 Cells
- Humans
- Liver Neoplasms -- drug therapy
- Mice
- Mice, Nude
- Promoter Regions, Genetic
- RNA Interference
- RNA, Small Interfering -- metabolism
- Transcription Factors, TFII -- metabolism
- Transcription, Genetic
- Unfolded Protein Response
- src-Family Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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