c-Jun N-terminal kinase mediated VEGFR2 sustained phosphorylation is critical for VEGFA-induced angiogenesis in vitro and in vivo. [electronic resource]
Producer: 20121231Description: 17-27 p. digitalISSN:- 1559-0283
- Animals
- Anthracenes -- pharmacology
- Cell Movement
- Cell Survival
- Cells, Cultured
- Dose-Response Relationship, Drug
- Enzyme Activation
- Human Umbilical Vein Endothelial Cells -- cytology
- Humans
- Indoles -- pharmacology
- JNK Mitogen-Activated Protein Kinases -- metabolism
- MAP Kinase Signaling System
- Mice
- Mice, Inbred C57BL
- Neovascularization, Physiologic
- Phosphorylation
- Pyrroles -- pharmacology
- RNA, Small Interfering -- metabolism
- Recombinant Proteins -- pharmacology
- Transfection
- Vascular Endothelial Growth Factor A -- pharmacology
- Vascular Endothelial Growth Factor Receptor-2 -- antagonists & inhibitors
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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