Limiting multiple sclerosis related axonopathy by blocking Nogo receptor and CRMP-2 phosphorylation. [electronic resource]
Producer: 20120727Description: 1794-818 p. digitalISSN:- 1460-2156
- Adult
- Analysis of Variance
- Animals
- Antibodies -- therapeutic use
- Axons -- metabolism
- CD3 Complex -- metabolism
- Cell Line, Tumor
- Demyelinating Diseases -- etiology
- Disease Models, Animal
- Encephalomyelitis, Autoimmune, Experimental -- complications
- Female
- GPI-Linked Proteins -- antagonists & inhibitors
- Gene Expression Regulation -- genetics
- Glycoproteins -- adverse effects
- Green Fluorescent Proteins -- genetics
- Humans
- Immunoprecipitation
- Intercellular Signaling Peptides and Proteins -- genetics
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Middle Aged
- Multiple Sclerosis -- complications
- Mutation -- genetics
- Myelin Proteins -- antagonists & inhibitors
- Myelin-Oligodendrocyte Glycoprotein
- Nerve Degeneration -- etiology
- Nerve Tissue Proteins -- genetics
- Neuroblastoma -- pathology
- Neurofilament Proteins -- metabolism
- Nogo Receptor 1
- Optic Nerve -- metabolism
- Peptide Fragments -- adverse effects
- Phosphorylation
- Receptors, Cell Surface -- antagonists & inhibitors
- Retinal Ganglion Cells -- metabolism
- Severity of Illness Index
- Silver Staining
- Spinal Cord -- metabolism
- Time Factors
- Transduction, Genetic
- Tubulin -- metabolism
- tau Proteins -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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