Integrating role of T antigen, Rb2/p130, CTCF and BORIS in mediating non-canonical endoplasmic reticulum-dependent death pathways triggered by chronic ER stress in mouse medulloblastoma. [electronic resource]
Producer: 20120917Description: 1841-50 p. digitalISSN:- 1551-4005
- Animals
- Antigens, Viral, Tumor -- metabolism
- Apoptosis
- CCCTC-Binding Factor
- Caspase 12 -- metabolism
- Cell Death
- Chromatin Immunoprecipitation
- DNA-Binding Proteins -- genetics
- Endoplasmic Reticulum -- metabolism
- Endoplasmic Reticulum Stress
- Enzyme Activation
- Gene Expression Regulation, Neoplastic
- Glucose -- metabolism
- Medulloblastoma -- genetics
- Mice
- Mice, Transgenic
- Repressor Proteins -- genetics
- Retinoblastoma-Like Protein p130 -- genetics
- Signal Transduction
- Tumor Cells, Cultured
- Tumor Microenvironment
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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