Inhibition of constitutively activated phosphoinositide 3-kinase/AKT pathway enhances antitumor activity of chemotherapeutic agents in breast cancer susceptibility gene 1-defective breast cancer cells. [electronic resource]
Producer: 20131118Description: 667-75 p. digitalISSN:- 1098-2744
- Antineoplastic Agents -- pharmacology
- BRCA1 Protein -- deficiency
- Breast Neoplasms -- drug therapy
- Caspase 3 -- genetics
- Caspase 7 -- genetics
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Female
- Humans
- MCF-7 Cells
- Mutation -- genetics
- PTEN Phosphohydrolase -- genetics
- Phosphatidylinositol 3-Kinases -- genetics
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Signal Transduction -- drug effects
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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