LMW-E/CDK2 deregulates acinar morphogenesis, induces tumorigenesis, and associates with the activated b-Raf-ERK1/2-mTOR pathway in breast cancer patients. [electronic resource]
Producer: 20120904Description: e1002538 p. digitalISSN:- 1553-7404
- Acinar Cells -- cytology
- Animals
- Benzenesulfonates -- pharmacology
- Breast Neoplasms -- genetics
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Cell Transformation, Neoplastic -- drug effects
- Cyclin E -- genetics
- Cyclin-Dependent Kinase 2 -- antagonists & inhibitors
- Female
- Gene Expression Regulation, Neoplastic
- Humans
- Kaplan-Meier Estimate
- MAP Kinase Signaling System -- genetics
- Mammary Glands, Animal -- metabolism
- Mice
- Mice, Nude
- Neoplasm Invasiveness -- genetics
- Niacinamide -- analogs & derivatives
- Phenylurea Compounds
- Prognosis
- Protein Isoforms -- genetics
- Proto-Oncogene Proteins B-raf -- antagonists & inhibitors
- Purines -- pharmacology
- Pyridines -- pharmacology
- Retrospective Studies
- Roscovitine
- Sirolimus -- pharmacology
- Sorafenib
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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