Endogenously released ACh and exogenous nicotine differentially facilitate long-term potentiation induction in the hippocampal CA1 region of mice. [electronic resource]
Producer: 20120910Description: 1381-95 p. digitalISSN:- 1460-9568
- Acetylcholine -- metabolism
- Aconitine -- analogs & derivatives
- Analysis of Variance
- Animals
- Animals, Newborn
- Biophysics
- CA1 Region, Hippocampal -- cytology
- Dihydro-beta-Erythroidine -- pharmacology
- Electric Stimulation
- In Vitro Techniques
- Long-Term Potentiation -- drug effects
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Neurons -- drug effects
- Nicotine -- pharmacology
- Nicotinic Agonists -- pharmacology
- Nicotinic Antagonists -- pharmacology
- Patch-Clamp Techniques
- Receptors, Nicotinic -- deficiency
- Synaptic Potentials -- drug effects
- alpha7 Nicotinic Acetylcholine Receptor
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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