Colocalization of inflammatory response with B7-h1 expression in human melanocytic lesions supports an adaptive resistance mechanism of immune escape. [electronic resource]
Producer: 20120928Description: 127ra37 p. digitalISSN:- 1946-6242
- Adaptive Immunity -- immunology
- Adolescent
- Adult
- Aged
- Aged, 80 and over
- B7-H1 Antigen -- genetics
- Child
- Demography
- Female
- Follow-Up Studies
- Gene Expression Regulation, Neoplastic
- Humans
- Inflammation -- complications
- Interferon-gamma -- immunology
- Laser Capture Microdissection
- Lymphocytes, Tumor-Infiltrating -- immunology
- Male
- Melanocytes -- immunology
- Melanoma -- complications
- Middle Aged
- Neoplasm Staging
- Reverse Transcriptase Polymerase Chain Reaction
- Skin Neoplasms -- complications
- Survival Analysis
- Tumor Escape -- immunology
- Young Adult
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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