Deficiency of ATP13A2 leads to lysosomal dysfunction, α-synuclein accumulation, and neurotoxicity. [electronic resource]
Producer: 20120518Description: 4240-6 p. digitalISSN:- 1529-2401
- Animals
- Cells, Cultured
- Cerebral Cortex -- cytology
- Dementia -- pathology
- Embryo, Mammalian
- Epidermal Growth Factor -- metabolism
- ErbB Receptors -- metabolism
- Fibroblasts -- metabolism
- Gene Expression Regulation -- genetics
- Green Fluorescent Proteins -- genetics
- Humans
- L-Lactate Dehydrogenase -- metabolism
- Leucine -- metabolism
- Lysosomal-Associated Membrane Protein 1 -- metabolism
- Lysosomes -- metabolism
- Male
- Mice
- Mice, Inbred C57BL
- Microtubule-Associated Proteins -- metabolism
- Mutation -- genetics
- Neurofilament Proteins -- metabolism
- Neurons -- drug effects
- Parkinsonian Disorders -- pathology
- Proton-Translocating ATPases -- deficiency
- RNA, Small Interfering -- genetics
- Statistics, Nonparametric
- Time Factors
- Transfection
- Tritium -- metabolism
- alpha-Synuclein -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
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