TrkA gene ablation in basal forebrain results in dysfunction of the cholinergic circuitry. [electronic resource]
Producer: 20120518Description: 4065-79 p. digitalISSN:- 1529-2401
- Amino Acids -- metabolism
- Analysis of Variance
- Animals
- Animals, Newborn
- Cell Count
- Cell Size
- Choline O-Acetyltransferase -- metabolism
- Cholinergic Neurons -- physiology
- Conditioning, Psychological -- physiology
- Dopamine and cAMP-Regulated Phosphoprotein 32 -- metabolism
- Embryo, Mammalian
- Fear -- physiology
- Homeodomain Proteins -- genetics
- Maze Learning -- physiology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Prosencephalon -- cytology
- Proteins -- genetics
- RNA, Untranslated
- Receptor, Nerve Growth Factor -- metabolism
- Receptor, trkA -- deficiency
- Recognition, Psychology -- physiology
- Signal Transduction -- genetics
- Silver Staining
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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