Myb overexpression overrides androgen depletion-induced cell cycle arrest and apoptosis in prostate cancer cells, and confers aggressive malignant traits: potential role in castration resistance. [electronic resource]
Producer: 20120917Description: 1149-57 p. digitalISSN:- 1460-2180
- Androgens -- deficiency
- Apoptosis
- Cell Cycle Checkpoints -- genetics
- Cell Line, Tumor
- Cell Movement
- Cell Proliferation
- Cyclin A1 -- metabolism
- Cyclin D1 -- metabolism
- Cyclin E -- metabolism
- Epithelial-Mesenchymal Transition
- Humans
- Male
- Neoplasm Invasiveness
- Oncogene Proteins -- metabolism
- Oncogene Proteins v-myb -- biosynthesis
- Orchiectomy
- Proliferating Cell Nuclear Antigen -- biosynthesis
- Prostate -- metabolism
- Prostate-Specific Antigen -- biosynthesis
- Prostatic Neoplasms -- genetics
- Pseudopodia
- RNA Interference
- RNA, Small Interfering
- Transcription, Genetic
- Up-Regulation
- bcl-2-Associated X Protein -- biosynthesis
- bcl-Associated Death Protein -- metabolism
- bcl-X Protein -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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