PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice. [electronic resource]
Producer: 20120706Description: 1306-15 p. digitalISSN:- 1558-8238
- Action Potentials
- Amino Acid Substitution
- Animals
- Cells, Cultured -- drug effects
- Ganglia, Spinal -- cytology
- Hyperalgesia -- enzymology
- Ion Channel Gating
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mutation, Missense
- NAV1.8 Voltage-Gated Sodium Channel
- Point Mutation
- Protein Kinase C-epsilon -- analysis
- Protein Processing, Post-Translational
- Rats
- Sensory Receptor Cells -- enzymology
- Sodium -- metabolism
- Sodium Channels -- analysis
- Stress, Mechanical
- Substrate Specificity
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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