BMPR-II deficiency elicits pro-proliferative and anti-apoptotic responses through the activation of TGFβ-TAK1-MAPK pathways in PAH. [electronic resource]
Producer: 20120921Description: 2548-58 p. digitalISSN:- 1460-2083
- Animals
- Apoptosis
- Bone Morphogenetic Protein Receptors, Type II -- metabolism
- Cell Proliferation
- Cells, Cultured
- Familial Primary Pulmonary Hypertension
- Hypertension, Pulmonary -- metabolism
- MAP Kinase Kinase Kinases -- metabolism
- Mice
- Pulmonary Artery -- metabolism
- Rats
- Signal Transduction
- Transforming Growth Factor beta -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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