Autophagy inhibition promotes defective neosynthesized proteins storage in ALIS, and induces redirection toward proteasome processing and MHCI-restricted presentation. [electronic resource]
Producer: 20130103Description: 350-63 p. digitalISSN:- 1554-8635
- Adaptor Proteins, Signal Transducing -- metabolism
- Antigen Presentation -- drug effects
- Autophagy -- drug effects
- Green Fluorescent Proteins -- metabolism
- HeLa Cells
- Histocompatibility Antigens Class I -- immunology
- Humans
- Inclusion Bodies -- drug effects
- Intracellular Signaling Peptides and Proteins
- Molecular Chaperones -- metabolism
- NF-E2-Related Factor 2 -- metabolism
- Proteasome Endopeptidase Complex -- metabolism
- Proteasome Inhibitors -- pharmacology
- Protein Biosynthesis -- drug effects
- Protein Folding -- drug effects
- Protein Processing, Post-Translational -- drug effects
- Protein Structure, Quaternary
- Proteins -- metabolism
- Proteolysis -- drug effects
- Puromycin -- pharmacology
- Recombinant Fusion Proteins -- metabolism
- Ribosomes -- metabolism
- Sequestosome-1 Protein
- Signal Transduction -- drug effects
- Substrate Specificity -- drug effects
- Transcription, Genetic -- drug effects
- Ubiquitin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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