Chronic receptor-mediated activation of Gi/o proteins alters basal t-tubular and sarcolemmal L-type Ca²⁺ channel activity through phosphatases in heart failure. [electronic resource]
Producer: 20120530Description: H1645-54 p. digitalISSN:- 1522-1539
- 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester -- pharmacology
- Adrenergic beta-Agonists -- pharmacology
- Algorithms
- Animals
- Blood Pressure -- drug effects
- Calcium Channel Agonists -- pharmacology
- Calcium Channels, L-Type -- drug effects
- Cyclic AMP-Dependent Protein Kinases -- metabolism
- GTP-Binding Protein alpha Subunits, Gi-Go -- drug effects
- Heart Failure -- diagnostic imaging
- Heart Rate -- drug effects
- Isoproterenol -- pharmacology
- Male
- Membrane Potentials -- drug effects
- Mice
- Mice, Inbred C57BL
- Microtubules -- drug effects
- Myocardium -- pathology
- Myocytes, Cardiac -- physiology
- Patch-Clamp Techniques
- Pertussis Toxin -- pharmacology
- Phosphoprotein Phosphatases -- antagonists & inhibitors
- Receptors, G-Protein-Coupled -- drug effects
- Sarcolemma -- drug effects
- Ultrasonography
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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