Electroacupuncture pretreatment attenuates cerebral ischemic injury through α7 nicotinic acetylcholine receptor-mediated inhibition of high-mobility group box 1 release in rats. [electronic resource]
Producer: 20120606Description: 24 p. digitalISSN:- 1742-2094
- Animals
- Brain Injuries -- prevention & control
- Bridged Bicyclo Compounds, Heterocyclic -- therapeutic use
- Bungarotoxins -- pharmacology
- Cells, Cultured
- Cerebral Cortex -- cytology
- Cerebral Infarction -- etiology
- Disease Models, Animal
- Electroacupuncture -- methods
- Embryo, Mammalian
- Gene Expression Regulation -- drug effects
- Glucose -- deficiency
- HMGB1 Protein -- metabolism
- Hypoxia -- therapy
- In Situ Nick-End Labeling
- Infarction, Middle Cerebral Artery -- complications
- Injections, Intraventricular
- L-Lactate Dehydrogenase -- metabolism
- Male
- Neurons -- drug effects
- Neuroprotective Agents -- therapeutic use
- Phosphopyruvate Hydratase -- metabolism
- Protein Binding -- drug effects
- Quinuclidines -- therapeutic use
- Rats
- Rats, Sprague-Dawley
- Receptors, Nicotinic -- metabolism
- Statistics, Nonparametric
- Tetrazolium Salts
- Thiazoles
- alpha7 Nicotinic Acetylcholine Receptor
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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