Aryl hydrocarbon receptor protects lung adenocarcinoma cells against cigarette sidestream smoke particulates-induced oxidative stress. [electronic resource]
Producer: 20120426Description: 293-301 p. digitalISSN:- 1096-0333
- Adenocarcinoma -- metabolism
- Aryl Hydrocarbon Hydroxylases -- genetics
- Cell Line, Tumor
- Cell Proliferation
- Cytochrome P-450 CYP1B1
- DNA Damage -- drug effects
- G1 Phase Cell Cycle Checkpoints -- drug effects
- Gene Expression Regulation, Neoplastic
- Gene Knockdown Techniques
- Glucuronosyltransferase -- genetics
- Humans
- Lung Neoplasms -- metabolism
- Oxidative Stress -- drug effects
- Reactive Oxygen Species -- metabolism
- Receptors, Aryl Hydrocarbon -- metabolism
- Resting Phase, Cell Cycle -- drug effects
- Smoke -- adverse effects
- Tobacco Smoke Pollution -- adverse effects
- Xenobiotics -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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